CALL FOR PAPERS Mitochondrial Metabolism Quantitative proteomic analysis reveals novel mitochondrial targets of estrogen deficiency in the aged female rat heart

نویسندگان

  • T. S. Lancaster
  • S. J. Jefferson
  • J. Craig Hunter
  • Veronica Lopez
  • J. E. Van Eyk
  • E. G. Lakatta
  • D. H. Korzick
چکیده

T. S. Lancaster, S. J. Jefferson, J. Craig Hunter, Veronica Lopez, J. E. Van Eyk, E. G. Lakatta, and D. H. Korzick Department of Kinesiology, The Pennsylvania State University, University Park, Pennsylvania; IGDP in Physiology, The Pennsylvania State University, University Park, Pennsylvania; Department of Medicine, Johns Hopkins University, Baltimore, Maryland; and Laboratory of Cardiovascular Science, Intramural Research Program, National Institute on Aging, National Institutes of Health, Baltimore, Maryland

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Quantitative proteomic analysis reveals novel mitochondrial targets of estrogen deficiency in the aged female rat heart.

The incidence of myocardial infarction rises sharply at menopause, implicating a potential role for estrogen (E(2)) loss in age-related increases in ischemic injury. We aimed to identify quantitative changes to the cardiac mitochondrial proteome of aging females, based on the hypothesis that E(2) deficiency exacerbates age-dependent disruptions in mitochondrial proteins. Mitochondria isolated f...

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Lancaster TS, Jefferson SJ, Korzick DH. Local delivery of a PKCε-activating peptide limits ischemia reperfusion injury in the aged female rat heart. Am J Physiol Regul Integr Comp Physiol 301: R1242–R1249, 2011. First published August 31, 2011; doi:10.1152/ajpregu.00851.2010.—Reduced efficacy of cardioprotective interventions in the aged female heart, including estrogen replacement, highlights ...

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Local delivery of a PKCε-activating peptide limits ischemia reperfusion injury in the aged female rat heart.

Reduced efficacy of cardioprotective interventions in the aged female heart, including estrogen replacement, highlights the need for alternative therapeutics to reduce myocardial ischemia-reperfusion (I/R) injury in postmenopausal women. Here, we sought to determine the efficacy of protein kinase-Cε (PKCε)-mediated cardioprotection in the aged, estradiol-deficient rat heart. Infarct size and fu...

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The effect of eight weeks of aerobic training with Propolis on some mitochondrial biogenesis markers in cardiac tissue of ovariectomized diabetic rats

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CALL FOR PAPERS Sex Diferences in Renal and Cardiovascular Function: Physiology and Pathophysiology Estrogen deficiency decreases ischemic tolerance in the aged rat heart: roles of PKC , PKC , Akt, and GSK3

Hunter JC, Kostyak JC, Novotny JL, Simpson AM, Korzick DH. Estrogen deficiency decreases ischemic tolerance in the aged rat heart: roles of PKC , PKC , Akt, and GSK3 . Am J Physiol Regul Integr Comp Physiol 292: R800–R809, 2007. First published September 28, 2006; doi:10.1152/ajpregu.00374.2006.—The mechanisms underlying the age-dependent reversal of female cardioprotection are poorly understoo...

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تاریخ انتشار 2012